张兆强，硕士研究生学历，教授，硕士研究生导师，现为毒理学教研室主任。
教学：承担《毒理学基础》、《职业卫生与职业医学》、《食品毒理学》等课程的教学工作。
科研：研究方向：矽肺纤维化发生机制。主持山东省自然科学基金、山东省卫健委、教育厅、济宁市、济宁医学院各层次课题8项；以第一作者在国内外期刊发表论文30余篇。
代表性课题：
1.山东省自然科学基金：巨噬细胞TLR4对矽肺纤维化枢纽因子TGFβ1调控的信号通路及医学干预研究，2017-2020，主持。
2.山东省自然科学基金：成纤维细胞中miR-207靶向调控TGF-β1/SMAD3对矽肺纤维化的抑制作用及机制研究，2022-2024，主持。
代表性论文：
1.Zhao-qiang Zhang, Chun-zhi Zhang, Bo Shao, et al. Effects of abnormal expression of fusion and fission genes on the morphology and function of lung macrophage mitochondria in SiO2-induced silicosis fibrosis in rats in vivo. Toxicology Letters. 2019,312:181-187.
2.Zhao-qiang Zhang, Bo Shao, Gui-zhi Han, et al. Location and dynamic changes of inflammation, fibrosis, and expression levels of related genes in SiO2-induced pulmonary fibrosis in rats in vivo. Journal of Toxicologic Pathology. 2019, 32(4):253-260.
3. Zhao-qiang Zhang, Hai-Tao Tian, Hu Liu, et al. The role of macrophage-derived TGF-β1 on SiO2- induced pulmonary fibrosis: A review. Toxicology and Industrial Health. 2021,37(4):240-250.
4.Zhao-qiang Zhang, Xiao Wu, Gui-zhi Han, Bo Shao, Lin L, Shun-li Jiang. Altered M1/M2 polarization of alveolar macrophages is involved in the pathological responses of acute silicosis in rats in vivo. Toxicol Ind Health. 2022,38(12):810-818.
5. Jia-hui Zhao, Shuang Li, Shu-ling Du, Zhao-qiang Zhang(通信作者). The role of mitochondrial dysfunction in macrophages on SiO2 -induced pulmonary fibrosis: A review. J Appl Toxicol. 2023 Jul 19. doi: 10.1002/jat.4517. Epub ahead of print.
6.Shuang Li, Jiahui Zhao, Guizhi Han, Xin Zhang, Ning Li, Zhaoqiang Zhang(通信作者). Silicon dioxide- induced endoplasmic reticulum stress of alveolar macrophages and its role on the formation of silicosis fibrosis: a review article. 2023,Oct 28. https://doi.org/10.1093/toxres/tfad099. Epub ahead of print.